Benjamin Lab Publications
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Selected Publications:
- Ralser M, Benjamin IJ. Reductive stress on life span extension in C. elegans. BMC Research Notes 1:19, 2008.
- Rajasekaran NS, Firpo MA*, Connell P, Milash B, Weiss R, Benjamin IJ. Gene Expression Profiling Identifies Glutathione Metabolic Upregulation as an Early Molecular Signature of R120G CryAB Cardiomyopathic Mice. Physiological Genomics. 2008, Jul 15.
- Reinke H, Saini C, Fleury-Olela F, Dibner C, Benjamin IJ, Schibler U: Differential display of DNA-binding proteins reveals heat-shock factor 1 as a circadian transcription factor. Genes Dev, 22(3), 331-45, 2008.
- Pinz I, Robbins J, Rajasekaran NS, Benjamin IJ, Ingwall JS. (2007). Unmasking different mechanical and energetic roles for the small heat shock proteins CryAB and HSPb2 using genetically modified mouse hearts. FASEB J, 22:84-92, 2008.
- Benjamin IJ, Guo Y, Srinivasan S, Boudina S, Taylor RP, Rajasekaran NS, Gottlieb R, Wawrousek EF, Abel ED, Bolli R: CRYAB and HSPB2 deficiency alters cardiac metabolism and paradoxically confers protection against myocardial ischemia in aging mice. AJP-Heart Circ Physiol. 293: H3201-3209, 2007.
- Rajasekaran NS, Connell P, Christians ES, Yan L-J, Taylor RP, Orosz A, Zhang XQ, Stevenson TJ, Peshock RM, Leopold JA, Barry WH, Loscalzo J, Odelberg SJ, and Benjamin IJ: Human alphaB-Crystallin Mutation Causes Oxido-reductive Stress and Protein Aggregation Cardiomyopathy in Mice. Cell,130(3), 427-39, 2007.
- Corey LL, Weirich CS, Benjamin IJ, Kingston RE: Localized recruitment of a chromatin-remodeling activity by an activator in vivo drives transcriptional elongation. Genes Dev Jun 1;17(11):1392-401, 2003.
- Yan L-J, Christians E, Liu L, Xiao X, Sohal S, and Benjamin IJ. Mouse Heat Shock Transcription Factor 1 Deficiency Alters Cardiac Redox Homeostasis and Increases Mitochondrial Oxidative Damage. EMBO J 21:5164-72, 2002.
- Christians E, Davis AD, Thomas S, and Benjamin IJ. Maternal effect of Hsf1 on Reproductive success. Nature, 407,(6805) 693-694, 2000.
- Xiao X, Davis AA, McMillan DR, Curry B, Richardson J, and Benjamin IJ. HSF1 is required for Extraembryonic Development, Postnatal Growth and Protection during Inflammatory Responses in Mice. EMBO J. Vol. 18: 5943‑5952, 1999.
