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Bellamkonda K. Kishore

Bellamkonda K. Kishore, MD, PhD, MBA, FASN, FRSB

Languages spoken: English, Telugu, Hindi, French

Academic Information

Departments Adjunct - Internal Medicine

Divisions: Nephrology

Academic Office Information

BK.Kishore@hsc.utah.edu

Research Interests

  • Pathophysiology of Acquired Nephrogenic Diabetes Insipidus
  • Role of Purinergic Signaling in Renal Physiology
  • Pathophysiology and Experimental Therapeutics
  • Diet-Induced Obesity and Insulin Resistance
  • Induction of Native Erythropoietin Production in the Kidney

Education History

Graduate Training University of Utah, David Eccles School of Business
Executive MBA
NHLBI, National Institutes of Health
Visiting Fellow
Fellowship Catholic University of Louvain, Science Development Foundation
Fellow
Catholic University of Louvain
PhD
Fellowship International Institute of Cellular and Molecular Pathology
Fellow
Niigata University School of Medicine, Second Department of Internal Medicine
Fellow
Professional Medical Banaras Hindu University
MD
Government General Hospital
Intern
Undergraduate Kurnool Medical College, SV University
MB, BS

Selected Publications

Journal Article

  1. Zhang Y, Peti-Peterdi J, Brandes AU, Riquier-Brison A, Carlson NG, Muller CE, Ecelbarger CM, Kishore BK (2017). Prasugrel suppresses development of lithium-induced nephrogenic diabetes insipidus in mice. Purinergic Signal, 13, 239-248.
  2. Zhang Y, Peti-Peterdi J, Heiney KM, Riquier-Brison A, Carlson NG, Muller CE, Ecelbarger CM, Kishore BK (2015). Clopidogrel attenuates lithium-induced alterations in renal water and sodium channels/transporters in mice. Purinergic Signal, 11(4), 507-18.
  3. Thimm D, Schiedel AC, Peti-Peterdi J, Kishore BK, Mller CE (2015). The nucleobase adenine as a signalling molecule in the kidney. Acta Physiol (Oxf), 213, 808-818.
  4. Kishore BK, Carlson NG, Ecelbarger CM, Kohan DE, Mller CE, Nelson RD, Peti-Pterdi J, Zhang Y (2015). Targeting renal purinergic signalling for the treatment of lithium-induced nephrogenic diabetes insipidus. Acta Physiol (Oxf), 214, 176-188.
  5. Zhang Y, Li L, Kohan DE, Ecelbarger CM, Kishore BK (2013). Attenuation of lithium-induced natriuresis and kaliuresis in P2Y2 receptor knockout mice. Am J Physiol Cell Physiol, 305, F407-F416.
  6. Kishore BK, Zhang Y, Geovrgyan H, Kohan DE, Schiedel AC, Muller CE, Peti-Peterdi J (2013). Cellular localization of adenine receptors (AdeR) in the rat kidney, and their functional significance in the inner medullary collecting duct. Aug 28, 1-8.
  7. Zhang Y, Pop IL, Carlson NG, Kishore BK (2012). Genetic deletion of P2Y2 receptor offers significant resistance for the development of lithium-induced polyuria accompanied with alterations in PGE2 signaling. 302, F70-F77.
  8. Zhang Y, Morris KM, Sparrow SK, Dwyer KM, Enjyoji K, Robson SC, Kishore BK (2012). Defective renal water handling in transgenic mice over-exprssing human CD39/NTPDase1. 303, F420-F430.
  9. Zhang Y, Listhrop R, Ecelbarger CM, Kishore BK (2011). Renal sodium transporter/channel expression and sodium excretion in P2Y2 receptor knockout mice fed high NaCl diet with/without aldosterone infusion. 300, F657-F668.
  10. Zhang Y, Kohan DE Nelson RD, Carlson NG, Kishore BK (2010). Potential involvement of P2Y2 receptor in diuresis of post-obstructive uropathy in rats. 298, F634-F642.
  11. Zhang Y, Nelson RD, Carlson NG, Kamerath CD, Kohan DE, Kishore BK (2009). Potential role of purinergic signaling in lithium-induced nephrogenic diabetes insipidus. 296, F1194-1201.
  12. Zhang Y, Sands JM, Kohan DE, Nelson RD, Martin CF, Carlson NG, Kamerath CD, Ge Y, Kelin JD, Kishore BK (2008). Potential role of purinergic signaling in urinary concentration in inner medulla: Insights from P2Y2 gene knockout mice. 295, F1715-F1724.
  13. Kishore BK, Isaac J, Fausther M, Tripp SR, Shi H, Gill PS, Braun N, Zimmermann H, Sevigny J, Robson SC (2005). Expression of NTPDase1 and NTPDase2 in murine kidney: relevance to regulation of P2 receptor signaling. Am J Physiol Renal Physiol, 288(5), F1032-43.

Review

  1. Kishore BK, Ecelbareger CM (2013). Lithium: A versatile tool for understanding renal physiology. [Review]. 304, F1139-F1149.
  2. Kishore BK, Nelson RD, Miller RL, Carlson NG, Kohan DE (2009). P2Y2 receptor and water transport in the kidney. [Review]. Purinergic Signal, 5, 491-499.
  3. Krane CM, Kishore BK (2003). Aquaporins: Membrane water channels of the biological world. [Review]. Biologist (London), 50, 81-86.
  4. Mingeot-Leclercq MP, Laurent G, Kishore BK, Tulkens PM (1991). Aminoglycoside Nephrotoxicity. [Review]. 10, 113-141.

Book Chapter

  1. Peti-Peterdi J, Kishore BK, Pluznick JL (2016). Regulation of Vascular and Renal Function by Metabolite Receptors. In Annual Review of Physiology (2016, 78, pp. 391-414).
  2. Kishore BK, Maldague P, Laurent G, Mingeot-Leclercq MP, Ibrahim S, Tulkens PM (1994). Acute renal failure induced by aminoglycoside antibiotics: Pathophysiology, clinical implications and development of protective measures. In Chugh KS (Ed.), Asian Nephrology (pp. 401-409). Delhi: Oxford University Press.

Patent

  1. Kishore B, Zhang Y, Carlson N (2019). Treatment of Kidney Diseases Associated with Elevated AVP Levels. U.S. Patent No. PCT/US2017/62/452,841 filed on 02/19/2017. Washington, D.C.:U.S. Patent and Trademark Office.
  2. Kishore B, Zhang Y, Ecelbarger C (2018). Composition and Methods for the Prevention and Treatment of Diet-induced Obesity. U.S. Patent No. 10,024,846. Washington, D.C.:U.S. Patent and Trademark Office.
  3. Kishore B, Carlson N, Kohan D, Nelson R (2018). Methods and Composition for Treating Nephrogenic Diabetes Insipidus. U.S. Patent No. 9,901,624. Washington, D.C.:U.S. Patent and Trademark Office.
  4. Kishore B, Carlson N, Zhang Y (2017). Methods and Composition for Treating Acquired Nephrogenic DIabetes Insipidus. U.S. Patent No. 9,539,246. Washington, D.C.:U.S. Patent and Trademark Office.
  5. Kishore B, Westenfelder C, Isaac J (2011). Composition and Methods Related to Production of Erythropoietin. U.S. Patent No. 8,084,423. Washington, D.C.:U.S. Patent and Trademark Office.